M.A.,Psychology University of North Carolina Wilmington, 2015
- Alzheimer’s Disease
- Glutamate Dysregulation
- Tau Phosphorylation
Sharay Setti research focuses on the underlying mechanisms of Alzheimer’s disease pathology and progression. Setti is particularly interested in the role of the lateral entorhinal cortex in early Alzheimer’s disease development. Setti’s goal is to understand that role, and examine ways it can rescue pathology by targeting that region specifically.
- Baumgarner, K. M., Setti, S., Diaz, C., Littlefield, A., Jones, A., & Kohman, R. A. (2014). Diet-induced obesity attenuates cytokine production following an immune challenge. Behavioural brain research, 267, 33-41.
- Setti, S., Littlefield, A., Johnson, S., Kohman, R. A. (2015). Diet-induced obesity prevents endotoxin-induced cognitive deficits. Physiology and Behavior
- Littlefield, A.M., Setti, S.E., Priester, C., Kohman, R.A. (2015). Voluntary exercise attenuates LPS-induced reductions in neurogenesis and increases microglia expression of a proneurogenic phenotype in aged mice. Journal of Neuroinflammation.
- Hunsberger, H.C., Wang, D., Petrisko, T.J., Alhowail, A., Setti, S.E., Suppiramaniam, V., Konat, G.W., Reed, M.N. (2016). Peripherally restricted viral challenge elevates extracellular glutamate and enhances synaptic transmission in the hippocampus. Journal of Neurochemistry.